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I’ve had at least 3 diagnosed under your requested age. We did not do stim test, just collection of blood. All were also IR. One was an Arabian, age 13 when diagnosed, and two are minis, diagnosed at age 9 and 13. Lost the Arab due to Cushings at age 21 and the minis are currently 20 y/o & 24. I also have 2 donkeys confirmed and on pergolide; one diagnosed at 22 (now 25) and one diagnosed at 16 (now 19).
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My Thoroughbred gelding was diagnosed at 12 from an ACTH blood draw. We checked insulin too, a few times, but that has been in the normal range. He is doing fine on a 1/2 tablet of Prascend every day. He started on 1 tablet for a month then went down to a 1/2 tablet. We checked every 3 months for a year and now, at age 14, my vet wants to retest him every April and October.
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With my horse diagnosed about 13 I do believe that diet may have played a part. I know this is up for discussion, but it fits with this horse.
Half arab pony on 1/4 pill and doing fine.
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Correlation != causation 
The entirety of the Equine Endocrinology Group says this isn’t caused by diet. ay I ask why you feel it played a role?
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Yea I know, but since we have to adjust the diet it makes me suspicious. And my horse was fed a very high nsc feed when I bought him.
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Very interesting. I JUST heard a vet on a podcast say they are questioning correlation between high insulin and later the development of PPID.
It was Van Epps on the Humble Hoof podcast. And he’s a world leader in research on laminitis.
I think it was this one.
And I’d been thinking for a few year now how important it is to KNOW and make sure our horses are not running high insulin. And know how hard that can be to manage because I’ve had IR Horses for, shit, 30 yrs? It’s a horrible diagnosis and then sure enough had two IR horses turn PPID.
Prascend is expensive and you know you’re inclined then to a total attack on their bodies and that is what I’ve witnessed. And somehow have not dealt with laminitis. And maybe we were on the verge at time and avoided disaster.
My current plan is to install a track around my property and get them off the grass, moving more and see what that does for insulin numbers. It’s a grand experiment. 
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that, or the other way around? It’s been known that PPID predisposes a horse to also becoming IR. Are you saying they’re thinking the horse is already, or borderline, EMS, and therefore more likely to become PPID, which then really predisposes the non-EMS but headed there horse, to become truly EMS?
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That a horse who is EMS is more likely to later become PPID.
Interesting! But, probably not all that surprising, since EMS can really affect the whole body. I wonder if there’s any connection to whether the pituitary is enlarging, or has a tumor. My gut on this says EMS might be more likely to cause enlargement over tumors?
Now I’m really curious about the % of horses with PPID in a given breed that is not predisposed to being IR (like TBs) vs the % in breeds who are.
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