According to research by Dr. Nicholas Dodman at Tufts College of Veterinary Medicine in 2000, fear-aggressive behavior was significantly reduced in dogs fed a low (17%) protein diet when compared to medium (25%) and high (32%) protein diets. The same study concluded that dietary protein had no effect on dominant-aggressive dogs._
these are dogs who are fearful. Already afraid. Not aggressive, extremely fearful. They are being TREATED by feeding a different diet. Protein didn’t CAUSE them to be this way- where does it say they became fearful after being fed a high-protein diet? They are probably fearful due to genetics and bad experiences. Note the actually aggressive dogs didn’t respond to the diet. If your dog isn’t already fear-aggressive then starving him of protein is ridiculous. And unhealthy. I note you didn’t post another study by Dodman where they concluded:
“Results of this study suggest that a reduction in dietary protein content is not generally useful in the treatment of behavior problems in dogs”
If you like I will dig up the endless number of studies demonstrating dogs need 30% or higher protein to be healthy. Especially older dogs.
See this report of a puppy almost crippled by malnutrition? it doesn’t give the brand name but I suspect the diet that crippled the puppy is Honest Kitchen’s Preference plus ground beef:
J Am Vet Med Assoc. 2009 Apr 15;234(8):1041-8.
Diffuse osteopenia and myelopathy in a puppy fed a diet composed of an organic premix and raw ground beef.
Taylor MB, Geiger DA, Saker KE, Larson MM.
SourceBanfield, The Pet Hospital, Christiansburg, VA 24060, USA.
Abstract
CASE DESCRIPTION: An 8-month-old Shetland Sheepdog was evaluated because of the sudden onset of signs of neck pain, collapse, and inability to rise. A cursory diet history indicated that the dog had been fed a raw meat-based diet.
CLINICAL FINDINGS: Initial evaluation of the dog revealed small physical stature, thin body condition, and signs of cranial cervical myelopathy. Radiographically, diffuse osteopenia of all skeletal regions was identified; polyostotic deformities associated with fracture remodeling were observed in weight-bearing bones, along with an apparent floating dental arcade. Hypocalcemia and hypophosphatemia were detected via serum biochemical analyses. The dog’s diet was imbalanced in macronutrients and macrominerals.
TREATMENT AND OUTCOME: The dog received supportive care and treatment of medical complications; neurologic abnormalities improved rapidly without intervention. Dietary changes were implemented during hospitalization, and a long-term feeding regimen was established. Following discharge from the hospital, exercise restriction was continued at home. Serial follow-up evaluations, including quantitative bone density measurements, revealed that dietary changes were effective. After 7 months, the dog was clinically normal.
CLINICAL RELEVANCE: In the dog of this report, vitamin D-dependent rickets type I and suspected nutritional secondary hyperparathyroidism developed following intake of a nutritionally incomplete and unbalanced diet. The raw meat-based, home-prepared diet fed to the dog was not feed-trial tested for any life stage by the Association of American Feed Control Officials, and its gross nutrient imbalance induced severe metabolic, orthopedic, and neurologic abnormalities. Inadvertent malnutrition can be avoided through proper diet assessment and by matching nutrient profiles with patients’ nutritional needs.